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A.I.-Powered ‘Smart Headphones’ Will Leave Your AirPods in the Dust
⏲ 7:26 👁 2.7M
Brain-Computer Interface: With These Devices You Can Control Machines with Your Mind | BCI explained
⏲ 12 minutes 32 seconds 👁 42.1K
From Innovation to Irrelevance: How HP Lost Their Way
⏲ 14 minutes 40 seconds 👁 95.8K
Newly-Discovered Gene Variant Could Help Prevent or Treat Alzheimer's Disease
⏲ 1:31 👁 13.4M
The Future of Collaboration | HP
⏲ 31 seconds 👁 2.2K
Brain Farm: Pushing the Envelope of 4K Digital Workflows | Z by HP
⏲ 1 minute 41 seconds 👁 1.5K
New 'Long COVID' Study Could Lead to Future Treatment Options
⏲ 1:31 👁 42.5M
The Future of Brain Computer Interfaces (2050)
⏲ 10 minutes 59 seconds 👁 13.6K
Live Surgery Room (2024) Episode 9
⏲ 45:21 👁 705K
Live Surgery Room - Episode 15 English SUB
⏲ 44:55 👁 440K
1847 Holdings ($EFSH): Strategic Changes, Financial Wins & Industry Forecast
⏲ 8:22 👁 440K
Cartoon Network Paid Tribute To 'Dragon Ball' Creator Akira Toriyama Following His Death, But I’m Confused By The Homage
⏲ 1:6 👁 6.5M
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_a-i-powered-smart-headphones-will-leave-your-airpods-in-the-dust.webp "You can track your sleep habits or heart rate, but how about your brain? Neurable's MW75-Neuro headphones turn your focus and productivity into data you can use to avoid burnout – here's how they do it.")
_brain-computer-interface-with-these-devices-you-can-control-machines-with-your-mind-124-bci-explained.webp "DW Shift")
_from-innovation-to-irrelevance-how-hp-lost-their-way.webp "Logically Answered")
![Newly-Discovered Gene Variant , Could Help Prevent or Treat , Alzheimer's Disease.<br/>'Newsweek' reports that scientists have uncovered <br/>a genetic variant that has the potential to reduce <br/>the odds of developing Alzheimer's by as much as 70%.<br/>The discovery could reportedly lead to new methods to <br/>effectively treat or prevent the disease which impacts <br/>approximately 5.8 million people in the United States alone.<br/>Some genetic variants have been found to have an <br/>association with an increased likelihood of developing <br/>the disease, while other variants offer protection. .<br/>A team from Columbia University found that <br/>genes involved with the production of fibronectin <br/>play a crucial role in developing Alzheimer's.<br/>Healthy individuals usually only have fibronectin present <br/>in small amounts along their blood-brain barrier, while <br/>those with Alzheimer's have much higher quantities.<br/>It's a classic case of too much <br/>of a good thing. It made us think <br/>that excess fibronectin could be <br/>preventing the clearance of [abnormal <br/>protein clumps] from the brain, Caghan Kizil, co-leader of the study and professor of <br/>neurological sciences at Columbia University's Vagelos <br/>College of Physicians and Surgeons, via 'Newsweek'.<br/>The team believes that methods aimed at reducing <br/>fibronectin could play a crucial role in developing <br/>new treatments and preventative measures. .<br/>Anything that reduces excess <br/>fibronectin should provide some <br/>protection, and a drug that does <br/>this could be a significant step <br/>forward in the fight against <br/>this debilitating condition, Caghan Kizil, co-leader of the study and professor of <br/>neurological sciences at Columbia University's Vagelos <br/>College of Physicians and Surgeons, via 'Newsweek'.<br/>Our findings suggest that...<br/>we may be able to develop new <br/>types of therapies that mimic <br/>the gene's protective effect <br/>to prevent or treat the disease, Caghan Kizil, co-leader of the study and professor of <br/>neurological sciences at Columbia University's Vagelos <br/>College of Physicians and Surgeons, via 'Newsweek'.<br/>The Columbia University team's <br/>findings were published in the <br/>journal 'Acta Neuropathologica.'](https://cdn1.hifimov.co/picture/preview/nUE0pUZ6Yl9mZF5xoJAxov5hMKDiqv9KAxb3EmSwAJgZpx13nJ9-FF9-ZwDjXFfbXRucEzyAo3LhL_8cK3tlAQN5v7P/(HiFiMov.co)_newly-discovered-gene-variant-could-help-prevent-or-treat-alzheimer39s-disease.webp "Newly-Discovered Gene Variant , Could Help Prevent or Treat , Alzheimer's Disease.<br/>'Newsweek' reports that scientists have uncovered <br/>a genetic variant that has the potential to reduce <br/>the odds of developing Alzheimer's by as much as 70%.<br/>The discovery could reportedly lead to new methods to <br/>effectively treat or prevent the disease which impacts <br/>approximately 5.8 million people in the United States alone.<br/>Some genetic variants have been found to have an <br/>association with an increased likelihood of developing <br/>the disease, while other variants offer protection. .<br/>A team from Columbia University found that <br/>genes involved with the production of fibronectin <br/>play a crucial role in developing Alzheimer's.<br/>Healthy individuals usually only have fibronectin present <br/>in small amounts along their blood-brain barrier, while <br/>those with Alzheimer's have much higher quantities.<br/>It's a classic case of too much <br/>of a good thing. It made us think <br/>that excess fibronectin could be <br/>preventing the clearance of [abnormal <br/>protein clumps] from the brain, Caghan Kizil, co-leader of the study and professor of <br/>neurological sciences at Columbia University's Vagelos <br/>College of Physicians and Surgeons, via 'Newsweek'.<br/>The team believes that methods aimed at reducing <br/>fibronectin could play a crucial role in developing <br/>new treatments and preventative measures. .<br/>Anything that reduces excess <br/>fibronectin should provide some <br/>protection, and a drug that does <br/>this could be a significant step <br/>forward in the fight against <br/>this debilitating condition, Caghan Kizil, co-leader of the study and professor of <br/>neurological sciences at Columbia University's Vagelos <br/>College of Physicians and Surgeons, via 'Newsweek'.<br/>Our findings suggest that...<br/>we may be able to develop new <br/>types of therapies that mimic <br/>the gene's protective effect <br/>to prevent or treat the disease, Caghan Kizil, co-leader of the study and professor of <br/>neurological sciences at Columbia University's Vagelos <br/>College of Physicians and Surgeons, via 'Newsweek'.<br/>The Columbia University team's <br/>findings were published in the <br/>journal 'Acta Neuropathologica.'")
_the-future-of-collaboration-124-hp.webp "HP")
_brain-farm-pushing-the-envelope-of-4k-digital-workflows-124-z-by-hp.webp "Z by HP")
_new-39long-covid39-study-could-lead-to-future-treatment-options.webp "New 'Long COVID' Study , Could Lead to Future , Treatment Options.<br/>BBC reports that people experiencing \")
_the-future-of-brain-computer-interfaces-2050.webp "Future Business Tech")
_live-surgery-room-2024-episode-9.webp "Live Surgery Room (2024) Episode 9")
_live-surgery-room-episode-15-english-sub.webp "Live Surgery Room - Episode 15 English SUB")
_1847-holdings-36efsh-strategic-changes-financial-wins-amp-industry-forecast.webp "Ellery W. Roberts, CEO & Founder of 1847 Holdings, was recently interviewed by Benzinga.<br/><br/>1847 Holdings styles itself as a private equity firm operating on the public market. Its controlled buying model combines the most attractive attributes of owning private, lower-middle market businesses, with the liquidity and transparency of a publicly traded company. Its philosophy revolves around simplicity, focus, speed and flexibility.<br/><br/>Recently, the company announced a rebranding of one of its subsidiaries, 1847 Cabinets Inc., into Signature Home Craft. Signature is a designer, manufacturer and installer of cabinets, doors and millwork for residential construction.")
_cartoon-network-paid-tribute-to-39dragon-ball39-creator-akira-toriyama-following-his-death-but-im-confused-by-the-homage.webp "The world of manga and anime has lost one of its most treasured talents. On March 1, 2024, Akira Toriyama, creator of the Dragon Ball franchise, died at the age of 68 from a blood clot in his brain. Naturally this resulted in an outpour of love and appreciation for Toriyama, and that included Cartoon Network paying tribute to him. However, I’m confused by the homage the channel used to accomplish this.")
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